Search results for "Histone deacetylase 5"

showing 10 items of 12 documents

HDAC5 Inhibitors as a Potential Treatment in Breast Cancer Affecting Very Young Women

2020

Background: Breast cancer in very young women (BCVY) defined as &lt

0301 basic medicineOncologyCancer Researchmedicine.medical_specialtyDose dependencelcsh:RC254-282ArticleLMK-23503 medical and health sciences0302 clinical medicineBreast cancerbreast cancerOlder patientsInternal medicinemedicineskin and connective tissue diseasesPathologicalHDAC5 inhibitorsHistone deacetylase 5young womenbusiness.industrylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.disease030104 developmental biologyOncologyApoptosisCell culture030220 oncology & carcinogenesishistone deacetylaseHistone deacetylasebusiness
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Properties of the yeast nuclear histone deacetylase.

1994

A nuclear histone deacetylase from yeast was partially purified and some of its characteristics were studied. Histone deacetylase activity was stimulated in vitro by high-mobility-group nonhistone chromatin proteins 1 and 2 and ubiquitin and inhibited by spermine and spermidine, whereas n-butyrate had no significant inhibitory effect. Like the mammalian enzyme, partially purified histone deacetylase from yeast was strongly inhibited by trichostatin A. However, in crude extract preparations the yeast enzyme was not inhibited and treatment with trichostatin in vivo did not show any effect, either on the histone acetylation level or on cell viability. At low ionic strength, the enzyme can be i…

Cell NucleusHistone deacetylase 5HDAC11ChemistryHistone deacetylase 2HDAC10Cell BiologySaccharomyces cerevisiaeHydroxamic AcidsBiochemistryHistone DeacetylasesSubstrate SpecificityHistone Deacetylase InhibitorsMolecular WeightTrichostatin ABiochemistrymedicineChromatography GelHistone deacetylase activityHistone deacetylaseMolecular Biologymedicine.drugDeacetylase activityResearch ArticleThe Biochemical journal
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The Histone Deacetylase Inhibitor JAHA Down-Regulates pERK and Global DNA Methylation in MDA-MB231 Breast Cancer Cells

2015

The histone deacetylase inhibitor N-1-(ferrocenyl)-N-8-hydroxyoctanediamide (JAHA) down-regulates extracellular-signal-regulated kinase (ERK) and its activated form in triple-negative MDA-MB231 breast cancer cells after 18 h and up to 30 h of treatment, and to a lesser extent AKT and phospho-AKT after 30 h and up to 48 h of treatment. Also, DNA methyltransferase 1 (DNMT1), 3b and, to a lesser extent, 3a, downstream ERK targets, were down-regulated already at 18 h with an increase up to 48 h of exposure. Methylation-sensitive restriction arbitrarily-primed (MeSAP) polymerase chain reaction (PCR) analysis confirmed the ability of JAHA to induce genome-wide DNA hypomethylation at 48 h of expos…

DNA methyltransferase (DNMT)medicine.drug_classDNA methyltransferaselcsh:TechnologymedicineGeneral Materials ScienceCancer epigeneticsSettore BIO/06 - Anatomia Comparata E Citologialcsh:Microscopyhistone deacetylase inhibitorlcsh:QC120-168.85QD0415Histone deacetylase 5lcsh:QH201-278.5extracellular-signal-regulated kinase (ERK)ChemistryHistone deacetylase 2lcsh:TCommunicationAKTHistone deacetylase inhibitorMolecular biologySettore BIO/18 - Geneticalcsh:TA1-2040DNA methylationDNMT1lcsh:Descriptive and experimental mechanicslcsh:Electrical engineering. Electronics. Nuclear engineeringlcsh:Engineering (General). Civil engineering (General)lcsh:TK1-9971DNA hypomethylationQD0241
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Characterization of the NAD-Dependent Human Histone Deacetylases Gene Sirtuin 1 and Its Implications on Aging and the Development of Malignant Diseas…

2006

Abstract A dysregulation of the tightly controlled equilibrium of acetylation and deacetylation plays a causative role in the generation as well as in the suppression of cancer. Histone acetylation modifiers are therefore gaining increasing attention as potential targets in the treatment of cancer. Sirtuin 1 (SIRT1) is a nicotinamide adenine dinucleotide (NAD+)-dependent deacetylase, which belongs to the silent information regulator 2 (Sir2) family of sirtuin histone deacetylases (HDACs). The yeast Sir2 protein and its mammalian derivatives play a central role in epigenetic gene silencing, DNA repair and recombination, cell-cycle, microtubule organization, and in the regulation of aging. We…

GeneticsHistone deacetylase 5biologyHDAC11Sirtuin 1Histone deacetylase 2ImmunologyCell BiologyHematologySAP30BiochemistryHDAC4biology.proteinHistone codeHistone deacetylaseBlood
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Histone Deacetylase Inhibitors in the Treatment of Hematological Malignancies and Solid Tumors

2010

The human genome is epigenetically organized through a series of modifications to the histone proteins that interact with the DNA. In cancer, many of the proteins that regulate these modifications can be altered in both function and expression. One example of this is the family of histone deacetylases (HDACs), which as their name implies remove acetyl groups from the histone proteins, allowing for more condensed nucleosomal structure. HDACs have increased expression in cancer and are also believed to promote carcinogenesis through the acetylation and interaction with key transcriptional regulators. Given this, small molecule histone deacetylases inhibitors have been identified and developed…

Health Toxicology and Mutagenesislcsh:Biotechnologylcsh:MedicineReview ArticleNeoplasmslcsh:TP248.13-248.65GeneticsAnimalsHumansCancer epigeneticsMolecular BiologyHistone deacetylase 5biologyHDAC11Histone deacetylase 2HDAC10lcsh:RGeneral MedicineHistone Deacetylase InhibitorsHistoneBiochemistryAcetylationHematologic Neoplasmsbiology.proteinCancer researchMolecular MedicineHistone deacetylaseBiotechnologyJournal of Biomedicine and Biotechnology
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Distinct Site Specificity of Two Pea Histone Deacetylase Complexes

2001

We report on the site specificity of two intact pea histone deacetylase complexes. HD1 deacetylates lysines 5 and 16 of H4 in the order K16 > K5, while in the case of H3 the preferred order is K4 >> K18 approximately K9. The specificity of the HD2 complex is markedly different. The preferred residues in H4 are K8 approximately K5 > K16, while in H3 deacetylation, the complex HD2 prefers sites 4 and 18. To obtain these results, we have used a novel procedure based on the SPOT technique, a method to synthesize peptides on membrane supports. Different sets of membranes with sequentially overlapping histone peptides containing acetylated lysines in the sites corresponding to all in vivo acetyla…

Histone deacetylase 5Histone deacetylase 2ChemistryHDAC11HDAC10PeasSAP30BiochemistryHistone DeacetylasesSubstrate SpecificityBiochemistryHistone H2AHistone deacetylase complexDeacetylase activityBiochemistry
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Characterization of pea histone deacetylases

1988

The present paper is the first report on histone deacetylases from plants. Three enzyme fractions with histone deacetylase activity (HD0, HD1 and HD2) have been partially purified from pea (Pisum sativum) embryonic axes. They deacetylate biologically acetylated chicken histones and, to a lesser extent, chemically acetylated histones, this being a criterion of their true histone deacetylase nature. The three enzymes are able to accept nucleosomes as substrates. HD1 is not inhibited by n-butyrate up to 50 mM, whereas HD0 and HD2 are only slightly inhibited, thereby establishing a clear difference to animal histone deacetylases. The three activities are inhibited by acetate, Cu(2+) and Zn(2+) …

Histone deacetylase 5Histone deacetylase 2HDAC11HDAC10Plant ScienceGeneral MedicineBiologySAP30BiochemistryHistone methyltransferaseGeneticsHistone deacetylase activityHistone deacetylaseAgronomy and Crop SciencePlant Molecular Biology
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Protein phosphatases and chromatin modifying complexes in the inflammatory cascade in acute pancreatitis

2010

Acute pancreatitis is an inflammation of the pancreas that may lead to systemic inflammatory response syndrome and death due to multiple organ failure. Acinar cells, together with leukocytes, trigger the inflammatory cascade in response to local damage of the pancreas. Amplification of the inflammatory cascade requires up-regulation of pro-inflammatory cytokines and this process is mediated not only by nuclear factor κB but also by chromatin modifying complexes and chromatin remodeling. Among the different families of histone acetyltransferases, the p300/CBP family seems to be particularly associated with the inflammatory process. cAMP activates gene expression via the cAMP-responsive eleme…

Histone deacetylase 5biologyHistone methyltransferaseHistone H2Abiology.proteinCancer researchHistone acetyltransferaseHistone deacetylaseTopic HighlightSAP30CREBChromatin remodeling
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Synthesis of hybrid anticancer agents based on kinase and histone deacetylase inhibitors

2014

Fragments based on the VEGFR2i Semaxanib (SU5416, (vascular endothelial growth factor receptor-2\ud inhibitor) and the HDACi (histone deacetylase inhibitor) SAHA (suberanilohydroxamic acid) have been\ud merged to form a range of low molecular weight dual action hybrids. Vindication of this approach is\ud provided by SAR, docking studies, in vitro cancer cell line and biochemical enzyme inhibition data as well\ud as in vivo Xenopus data for the lead molecule (Z)-N1-(3-((1H-pyrrol-2-yl)methylene)-2-oxoindolin-5-yl)-\ud N8-hydroxyoctanediamide 6.

PharmacologyHistone deacetylase 5medicine.drug_classKinaseHistone deacetylase 2Organic ChemistryHistone deacetylase inhibitorQPharmaceutical ScienceBiologyBiochemistryHDACiVEGFRiHybrids.BiochemistryDocking (molecular)In vivoDrug DiscoverymedicineMolecular MedicineHistone deacetylaseSettore BIO/06 - Anatomia Comparata E CitologiaSemaxanibmedicine.drug
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The MAPK Hog1 recruits Rpd3 histone deacetylase to activate osmoresponsive genes

2003

Regulation of gene expression by mitogen-activated protein kinases (MAPKs) is essential for proper cell adaptation to extracellular stimuli. Exposure of yeast cells to high osmolarity results in rapid activation of the MAPK Hog1, which coordinates the transcriptional programme required for cell survival on osmostress. The mechanisms by which Hog1 and MAPKs in general regulate gene expression are not completely understood, although Hog1 can modify some transcription factors. Here we propose that Hog1 induces gene expression by a mechanism that involves recruiting a specific histone deacetylase complex to the promoters of genes regulated by osmostress. Cells lacking the Rpd3-Sin3 histone deac…

Saccharomyces cerevisiae ProteinsGenes FungalSaccharomyces cerevisiaeBiologySAP30Histone DeacetylasesOsmotic PressureGene Expression Regulation FungalPromoter Regions GeneticOligonucleotide Array Sequence AnalysisHistone deacetylase 5MultidisciplinaryHistone deacetylase 2HDAC11HDAC10HDAC9Molecular biologyHDAC4Cell biologyRepressor ProteinsMutationHistone deacetylase complexRNA Polymerase IIMitogen-Activated Protein KinasesProtein BindingTranscription FactorsNature
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